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Hypertrophic Pyloric Stenosis

0 Frequency: 0.1-0.4%. It is 15 times more common in those with a family history.

0 More than 50% of patients are the first child. It is 4 times more common in males.

Clinic and Diagnosis

• 3-6 of your life. Non-bilious Projectile vomiting is typical.

• The baby is always hungry, so he cries constantly and sucks the bottle hungrily while feeding.

• There may be hematemesis due to esophagitis.

• Subcutaneous adipose tissue decreased in patients due to nutritional deficiency.

• Mucous membranes due to dehydration are dry and skin turgor-tonus is decreased, constipation occurs, urine is decreased and dark.

• Severe hypoglycemia may occur because the liver stores are empty.

• Indirect bilirubin may increase due to low glucuronyl transferase activity in patients.

• Metabolic alkalosis due to vomiting occurs (hypokalemic, hypochloremic alkalosis).

• Palpation of "olive" on physical examination is pathognomonic.

• Other physical examination findings are increased gastric peristalsis and decreased skin turgor-ton.

• In Standing direct abdominal radiographs, there is a single and large gastric gas chamber, "bugi bugi" or "caterpillar" appearance due to contractions in the greater curvature.

• Diagnosis can be made by ultrasonography (bull's eye, doughnut, target sign).

• Radiogram with opaque material shows "railway" or "rope sign".

• Pyloric atresia, duplications, duodenal atresia, hiatal hernias, gastroenteritis.

• Rectal biopsy may be required for the differential diagnosis of Hirschsprung in the case of an episode of intestinal obstruction in the neonatal period.

Treatment

• First, the fluid-electrolyte imbalance is corrected.

• Fredet-Ramstedt pyloromyotomy is performed.

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